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Models of Alzheimer's disease propose a sequence of amyloid β (Aβ) accumulation, hypometabolism, and structural decline that precedes the onset of clinical dementia. These pathological features evolve both temporally and spatially in the brain. In this study, we aimed to characterise where in the brain and when in the course of the disease neuroimaging biomarkers become abnormal.

More information Original publication

DOI

https://doi.org/10.1016/S1474-4422(18)30028-0

Type

Journal article

Journal

The Lancet Neurology

Issue

3

Publisher

Elsevier

Publication Date

03/2018

Volume

17